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Upregulation of CHOP-10 (gadd153) expression in the mouse blastocyst as a response to stress

Published in: Molecular Reproduction and Development

Date of publication: December 1999

Language: English

Bibliographic databases:

Cites articles 30

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CHOP, a novel developmentally regulated nuclear protein that dimerizes with transcription factors C/EBP and LAP and functions as a dominant-negative inhibitor of gene transcription

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Apoptosis during retinoic acid-induced differentiation of F9 embryonal carcinoma cells
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Analysis of ATF3, a transcription factor induced by physiological stresses and modulated by gadd153/Chop10
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Physical and functional association between GADD153 and CCAAT/enhancer-binding protein beta during cellular stress
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Stress-induced binding of the transcriptional factor CHOP to a novel DNA control element

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Signals from the stressed endoplasmic reticulum induce C/EBP-homologous protein (CHOP/GADD153).
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A pathway distinct from the mammalian unfolded protein response regulates expression of endoplasmic reticulum chaperones in non-stressed cells
November 1, 1997

The growth arrest genes gas5, gas6, and CHOP-10 (gadd153) are expressed in the mouse preimplantation embryo
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Effects of nutrient deprivation and differentiation on the expression of growth-arrest genes (gas and gadd) in F9 embryonal carcinoma cells
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Expression and regulation of genes associated with cell death during murine preimplantation embryo development
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Amino acids and vitamins prevent culture-induced metabolic perturbations and associated loss of viability of mouse blastocysts
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Identification of novel stress-induced genes downstream of chop

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CHOP is implicated in programmed cell death in response to impaired function of the endoplasmic reticulum

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Hyperglycemia induces apoptosis in pre-implantation embryos through cell death effector pathways

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