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Thrombin-activatable fibrinolysis inhibitor (TAFI) deficiency is compatible with murine life

Published in: Journal of Clinical Investigation

Date of publication: January 1, 2002

Language: English

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Cites articles 58

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Title

Authors

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Interaction of one-chain and two-chain tissue plasminogen activator with intact and plasmin-degraded fibrin
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Optimized microturbidimetric assay for fibrinogen
August 1, 1989

An arginine specific carboxypeptidase generated in blood during coagulation or inflammation which is unrelated to carboxypeptidase N or its subunits
May 1, 1989

Characterisation of a carboxypeptidase in human serum distinct from carboxypeptidase N
July 1, 1989

Identification of the domains of tissue-type plasminogen activator involved in the augmented binding to fibrin after limited digestion with plasmin
June 1, 1989

Kaolin-induced writhing in mice, a new model of possible bradykinin-induced pain for assessment of analgesic agents
April 1, 1990

Antithrombotic effect of recombinant human thrombomodulin on thrombin-induced thromboembolism in mice
November 15, 1991

Isolation, molecular cloning, and partial characterization of a novel carboxypeptidase B from human plasma
October 1, 1991

The plasminogen activator/plasmin system
September 1, 1991

Evidence for a role of bradykinin in experimental pain models
October 1, 1992

The gene encoding human plasma carboxypeptidase B (CPB2) resides on chromosome 13
December 1, 1992

Oxidation of a specific methionine in thrombomodulin by activated neutrophil products blocks cofactor activity. A potential rapid mechanism for modulation of coagulation
December 1, 1992

The mechanism of plasminogen activation and the variability of the fibrin effector during tissue-type plasminogen activator-mediated fibrinolysis,The Mechanism of Plasminogen Activation and the Variability of the Fibrin Effector during Tissue‐type Plasminogen Activator—mediated Fibrinolysis
December 1, 1992

The intrinsic lysis concept,The Intrinsic Lysis Concepta
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Plasminogen activator inhibitor-1 gene-deficient mice. II. Effects on hemostasis, thrombosis, and thrombolysis
June 1, 1994

Carboxypeptidase U, a plasma carboxypeptidase with high affinity for plasminogen
1994

Pro-carboxypeptidase R cleaves bradykinin following activation
March 1, 1994

Physiological consequences of loss of plasminogen activator gene function in mice
July 1, 1995

The fibrinolytic system: from Petri dishes to genetic engineering
June 1, 1995

Purification and characterization of TAFI, a thrombin-activable fibrinolysis inhibitor
May 2, 1995

Activation and characterization of procarboxypeptidase B from human plasma
November 1995

Plasma carboxypeptidases as regulators of the plasminogen system
April 1, 1995

Plasminogen deficiency causes severe thrombosis but is compatible with development and reproduction
December 1, 1996

The gene for human carboxypeptidase U (CPU)--a proposed novel regulator of plasminogen activation--maps to 13q14.11.
November 1, 1996

Coagulation-dependent inhibition of fibrinolysis: role of carboxypeptidase-U and the premature lysis of clots from hemophilic plasma
July 12, 1996

TAFI, or plasma procarboxypeptidase B, couples the coagulation and fibrinolytic cascades through the thrombin-thrombomodulin complex
June 1, 1996

Fibrin deposition in tissues from endotoxin-treated mice correlates with decreases in the expression of urokinase-type but not tissue-type plasminogen activator
March 1, 1996

Impaired wound healing in mice with a disrupted plasminogen gene
August 1, 1997

Tissue factor pathway inhibitor gene disruption produces intrauterine lethality in mice
May 1, 1997

Thrombin-mediated activation of factor XI results in a thrombin-activatable fibrinolysis inhibitor-dependent inhibition of fibrinolysis
April 1, 1997

The plasma carboxypeptidases and the regulation of the plasminogen system,The Plasma Carboxypeptidases and the Regulation of the Plasminogen System
May 1, 1997

On the mechanism of the antifibrinolytic activity of plasma carboxypeptidase B

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