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The Werner syndrome RECQ helicase targets G4 DNA in human cells to modulate transcription.

Author

Lucas T. Graybuck

published in: ヒューマン・モレキュラー・ジェネティクス

date of publication: 2016-03-16

language: 英語

main subject: ウェルナー症候群

Bibliographic databases:

Cites articles 70

Date

Title

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Werner's syndrome a review of its symptomatology, natural history, pathologic features, genetics and relationship to the natural aging process
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A Review of its Symptomatology, Natural History, Pathologic Features, Genetics And Relationship to the Natural Aging Process
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Replicative life-span of cultivated human cells. Effects of donor's age, tissue, and genotype
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Protein synthetic errors do not increase during aging of cultured human fibroblasts
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Positional cloning of the Werner's syndrome gene
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The Bloom's syndrome helicase unwinds G4 DNA
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Human werner syndrome DNA helicase unwinds tetrahelical structures of the fragile X syndrome repeat sequence d(CGG)n
2001-07-01

The Bloom's and Werner's syndrome proteins are DNA structure-specific helicases
2001-04-01

Telomerase expression prevents replicative senescence but does not fully reset mRNA expression patterns in Werner syndrome cell strains
2001-02-08

Interactions between the Werner syndrome helicase and DNA polymerase delta specifically facilitate copying of tetraplex and hairpin structures of the d(CGG)n trinucleotide repeat sequence
2003-10-03

Gene expression profiling in Werner syndrome closely resembles that of normal aging

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2004-06-18

The Werner syndrome helicase and exonuclease cooperate to resolve telomeric D loops in a manner regulated by TRF1 and TRF2
2004-12-01

Defective telomere lagging strand synthesis in cells lacking WRN helicase activity.
2005-11-01

Elevated telomere-telomere recombination in WRN-deficient, telomere dysfunctional cells promotes escape from senescence and engagement of the ALT pathway

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Gene expression and DNA repair in progeroid syndromes and human aging

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POT1 stimulates RecQ helicases WRN and BLM to unwind telomeric DNA substrates

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DNA architecture: from G to Z.
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Human telomere, oncogenic promoter and 5'-UTR G-quadruplexes: diverse higher order DNA and RNA targets for cancer therapeutics
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Telomere dysfunction as a cause of genomic instability in Werner syndrome
2008-03

WRN controls formation of extrachromosomal telomeric circles and is required for TRF2DeltaB-mediated telomere shortening
2009-12-04

Altered gene expression in the Werner and Bloom syndromes is associated with sequences having G-quadruplex forming potential
2010-06-16

The Werner syndrome protein suppresses telomeric instability caused by chromium (VI) induced DNA replication stress

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Inflammatory networks during cellular senescence: causes and consequences
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Pyrimidine pool imbalance induced by BLM helicase deficiency contributes to genetic instability in Bloom syndrome
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Diagnostic microRNAs in myelodysplastic syndrome
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Aging, cellular senescence, and cancer

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