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Capillary Flow in the Brain Cortex During Changes in Oxygen Supply and State of Activation

Published in: Ciba Foundation Symposium

Date of publication: March 1, 1978

Language: English

Main subject: cerebral circulation

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Cites articles 15

Date

Title

Authors

May 1, 1919

The rate of diffusion of gases through animal tissues, with some remarks on the coefficient of invasion
May 1, 1919

The number and distribution of capillaries in muscles with calculations of the oxygen pressure head necessary for supplying the tissue
June 1, 1970

[Quantitative determination of local blood flow using hydrogen electrochemically produced in the tissue]
September 1, 1971

Intracellular redox changes in functioning cerebral cortex. II. Effects of direct cortical stimulation
July 1, 1972

Cerebral blood flow: its measurement and regulation
September 1, 1972

Cerebral pO 2 , pCO 2 and pH: changes during convulsive activity and their significance for spontaneous arrest of seizures
January 1, 1973

Polarographic measurements of the oxygen pressure performed simultaneously with optical measurements of the redox state of the respiratory chain in suspensions of mitochondria under steady-state conditions at low oxygen tensions
April 1, 1974

Determination of local blood flow (microflow) by electrochemically generated hydrogen. Construction and application of the measuring probe
December 1, 1975
August 1, 1975

Regulation of local tissuePo 2of the brain cortex at different arterial O2 pressures
March 1, 1975

Po2 of cat cerebral cortex: Response to breathing N2 and 100% O2
October 1, 1976

In vivo measurement of pyridine nucleotide fluorescence from cat brain cortex
October 1, 1976

Behavior of microflow and local PO2 of the brain cortex during and after direct electrical stimulation. A contribution to the problem of metabolic regulation of microcirculation in the brain
January 1, 1976

Local tissue PO2 and microflow of the brain cortex under varying arterial oxygen pressure
May 1, 1978

PO2 and microflow histograms of the beating heart in response to changes in arterial pO2

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