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Complement in acute inflammation

Published in: Journal of Pathology

Date of publication: February 1, 1969

Language: English

Main subject: inflammation, complement system proteins

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Cites articles 20

Date

Title

Authors

October 1, 1955

Inhibition of immune hemolysis by diisopropyl fluorophosphate
August 1, 1958

Observations on the treatment of rheumatic fever with salicylate, ACTH and cortisone. II. Combined salicylate and corticoid therapy and attempts at rebound suppression
January 1, 1959

The demonstration of the role of mediators in turpentine pleurisy in rats by experimental suppression of the inflammatory changes
July 1, 1959

Experimental suppression of the acute inflammatory changes of thermal injury
January 1, 1960

The suppression by antiesterases of increased capillary permeability in acute inflammation
November 1, 1963

COMPLEMENT AS A MEDIATOR OF INFLAMMATION. ENHANCEMENT OF VASCULAR PERMEABILITY BY PURIFIED HUMAN C'1 ESTERASE
August 1, 1964

LYSOLECITHIN AND THE ACTION OF COMPLEMENT
April 1, 1964

A LYMPH-NODE PERMEABILITY FACTOR IN THE TUBERCULIN REACTION.
July 1, 1965

A ROLE OF POLYMORPHONUCLEAR LEUKOCYTES AND COMPLEMENT IN NEPHROTOXIC NEPHRITIS
July 1, 1965

PATHOGENIC FACTORS IN VASULAR LESIONS OF EXPERIMENTAL SERUM SICKNESS
February 1, 1965

BOUND COMPLEMENT AND IMMUNOLOGIC INJURY OF BLOOD VESSELS
October 1, 1966

Nature and mode of action of antilymphocytic antiserum
August 1, 1966

The chemosuppression of chemotaxis
March 1, 1967

Anaphylatoxin in its relation to the complement system
May 1, 1967

Complement as a mediator of inflammation. II. Biological properties of anaphylatoxin prepared with purified components of human complement
February 1, 1967

Formation and functional significance of a molecular complex derived from the second and the fourth component of human complement
February 1, 1967

Central and peripheral effects of anti-lymphocyte sera
August 1, 1967

A plasmin-split fragment of C'3 as a new chemotactic factor
January 1, 1968

An electron-microscope study of the vascular response to mild thermal injury in the rat.
September 1, 1968

Inflammation in Agranulocytotic Rats

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