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Microtubule-active drugs suppress the closure of the permeability transition pore in tumour mitochondria

published in: FEBS Letters

date of publication: 1996-09-01

language: English

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Cites articles 12

Date

Title

Authors

1973-01-01

Interaction of calcium with mitochondria isolated from Ehrlich ascites tumour cells
1978-08-01

Association of mitochondria with microtubules in cultured cells
1980-06-01

Allosteric inhibition of the Ca2+-activated hydrophilic channel of the mitochondrial inner membrane by nucleotides
1981-12-01

Calcium transport by Ehrlich ascites cell mitochondria in vitro and in situ,Calcium transport by ehrlich ascites cell mitochondria in vitro and in situ
1983-07-01

Evidence for tubulin-binding sites on cellular membranes: plasma membranes, mitochondrial membranes, and secretory granule membranes
1984-03-01

Isolation of mitochondria from ascites tumor cells permeabilized with digitonin
1989-01-01

Calcium ion transport in mitochondria
1993-10-01

Spermine inhibition of the permeability transition of isolated rat liver mitochondria: an investigation of mechanism
1994-02-01

The Ca(2+)-induced permeability transition pore is involved in Ca(2+)-induced mitochondrial oscillations. A study on permeabilised Ehrlich ascites tumour cells
1994-07-01

Mitochondrial calcium spiking: a transduction mechanism based on calcium-induced permeability transition involved in cell calcium signalling
1994-02-01

Interactions between brain mitochondria and cytoskeleton: evidence for specialized outer membrane domains involved in the association of cytoskeleton-associated proteins to mitochondria in situ and in vitro
1995-04-01

Contribution of the mitochondrial permeability transition to lethal injury after exposure of hepatocytes to t-butylhydroperoxide

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