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Physiological changes in the bloodstream of chick embryos exposed to teratogenic doses of hypoxia

Published in: Developmental Biology

Date of publication: April 1, 1966

Language: English

Main subject: chicken embryo, cell biology

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Cites articles 11

Date

Title

Authors

September 1, 1951

Carbohydrate metabolism in insulin-treated chick embryos
August 1, 1952

Development of the excretory system
April 1, 1953

Some effects of NaC1 concentration on the development of early chick blastoderms in culture
December 1, 1957

Ontogenetic changes in the freezing point and sodium and potassium content of the subgerminal fluid and blood plasma of the chick embryo
March 1, 1959

Dinitrophenolinduced cataracts in the chick embryo
October 1, 1961

Lactic acid accumulation as a cause of hypoxia-induced malformations in the chick embryo,Lactic Acid Accumulation as a Cause of Hypoxia-Induced Malformations in the Chick Embryo
July 1, 1961

A quantitative study of the lethal and teratogenic effects of hypoxia on the three-day chick embryo,A quantitative study of the lethal and teratogenic effects of hypoxia on the three‐day chick embryo
November 1, 1963

TERATOGENIC SIGNIFICANCE OF IONIC AND FLUID IMBALANCES
January 1, 1964

Biological Aspects of the Yolk of the Hen's Egg
December 1, 1964

THE ETIOLOGY OF HYPOXIA-INDUCED MALFORMATIONS IN THE CHICK EMBRYO
April 1, 1966

Teratogenic effects of calcium salts on chick embryos

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