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Lamin B1 is required for mouse development and nuclear integrity

forfatter

Laurent Vergnes

Stephen G. Young

1952 -

Karen Reue

udgivet i: Proceedings of the National Academy of Sciences

dato for udgivelse: 2004-07-01

sprog: engelsk

Cites articles 39

Date

Titel

Forfattere

2004-02-01

Defects in nuclear structure and function promote dilated cardiomyopathy in lamin A/C-deficient mice
2003-08-09

LMNA mutations in atypical Werner's syndrome
2003-05-15

Recurrent de novo point mutations in lamin A cause Hutchinson-Gilford progeria syndrome

Francis Collins, Michael Boehnke
2003-06-01

The laminopathies: nuclear structure meets disease
2003-04-17

Lamin a truncation in Hutchinson-Gilford progeria.

Annachiara De Sandre-Giovannoli, Arnold Munnich, Stanislas Lyonnet, Martine Le Merrer, Claire L Navarro, Jeanne Amiel, Nicolas Levy
2003-04-01

Nuclear envelope proteins and neuromuscular diseases
2003-01-01

BayGenomics: a resource of insertional mutations in mouse embryonic stem cells

Patricia Babbitt, Thomas E Ferrin
2003-05-01

A progeroid syndrome in mice is caused by defects in A-type lamins
2002-09

Craniosynostosis in transgenic mice overexpressing Nell-1

Shun' Ichi Kuroda, Steve Miao
2002-02-18

Alteration of nuclear lamin organization inhibits RNA polymerase II-dependent transcription
2002-03

Homozygous defects in LMNA, encoding lamin A/C nuclear-envelope proteins, cause autosomal recessive axonal neuropathy in human (Charcot-Marie-Tooth disorder type 2) and mouse

Pierre Szepetowski, Annachiara De Sandre-Giovannoli
2002-03-01

Nuclear lamins: building blocks of nuclear architecture

Yosef Gruenbaum
2002-12-01

A cluster of eight hydroxysteroid dehydrogenase genes belonging to the aldo-keto reductase supergene family on mouse chromosome 13

Laurent Vergnes, Jack Phan, Karen Reue
2002-08

Life at the edge: the nuclear envelope and human disease
2002-11-01

Lamins: building blocks or regulators of gene expression?
2002-09-16

Zmpste24 deficiency in mice causes spontaneous bone fractures, muscle weakness, and a prelamin A processing defect

Stephen G. Young
2002-08-01

The IIIc alternative of Fgfr2 is a positive regulator of bone formation
2002-04-01

Defective prelamin A processing and muscular and adipocyte alterations in Zmpste24 metalloproteinase-deficient mice.

Annika Wernerson, Carlos López Otín, Zhongjun Zhou, Kjell Hultenby, Alberto M. Pendas, Karl Tryggvason
2002-01-01

Hutchinson-Gilford progeria syndrome: a pathologic study
2002-02-01

Edible vaccines: not quite ready for prime time
2001-12-01

Identification of essential genes in cultured mammalian cells using small interfering RNAs

Thomas Tuschl, Sayda M Elbashir
2001-12-01

The pathogenesis of craniosynostosis in the fetus
2001-03-01

Lamins and disease: insights into nuclear infrastructure
2001-01-01

Transcriptional repression, apoptosis, human disease and the functional evolution of the nuclear lamina
2000-12-11

Nuclear lamins A and B1: different pathways of assembly during nuclear envelope formation in living cells
2000-12-01

Head and/or CaaX domain deletions of lamin proteins disrupt preformed lamin A and C but not lamin B structure in mammalian cells
1999-11-29

Loss of A-type lamin expression compromises nuclear envelope integrity leading to muscular dystrophy
1999-12-01

Colocalization of intranuclear lamin foci with RNA splicing factors
1998-11-01

Aryl-hydrocarbon receptor is an inhibitory regulator of lipid synthesis and of commitment to adipogenesis
1998-01-01

Nuclear lamins: their structure, assembly, and interactions

Ueli Aebi
1997-06

A- and B-type lamins are differentially expressed in normal human tissues
1997-03-01

Rapid sequence analysis of gene trap integrations to generate a resource of insertional mutations in mice

Artikel - wd:Q24564213

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