Inventaire - your friends and communities are your best library - Inventaire

Tau reduction prevents Abeta-induced defects in axonal transport.

author

Kai Zhang

Bianxiao Cui

Lennart Mucke

published in: Science

date of publication: 2010-09-09

language: English

main subject: Microtubule-associated protein tau, Amyloid beta (A4) precursor protein

Cites articles 12

Date

Title

Authors

2010-07-22

Dendritic function of tau mediates amyloid-beta toxicity in Alzheimer's disease mouse models

Lars M. Ittner, Yazi D Ke, Fabien Delerue, Janet van Eersel, Anne Eckert, Jürgen Götz
2010-04-29

Loss of tau elicits axonal degeneration in a mouse model of Alzheimer's disease
2010-07-01

Amyloid-beta peptide oligomers disrupt axonal transport through an NMDA receptor-dependent mechanism that is mediated by glycogen synthase kinase 3beta in primary cultured hippocampal neurons

Michael A. Silverman, Karen Lo, Sérgio Teixeira Ferreira
2009-04-07

Disruption of fast axonal transport is a pathogenic mechanism for intraneuronal amyloid beta
2008-01-17

Differential regulation of dynein and kinesin motor proteins by tau

Jennifer L. Ross, Ram Dixit, Yale E. Goldman, Erika Holzbaur
2008-02-01

Neurotrophic factors in Alzheimer's disease: role of axonal transport

Katharina Schindowski, Karim Bélarbi, Luc Buée
2008-02-01

Axonal transport rates in vivo are unaffected by tau deletion or overexpression in mice
2007-05-01

Reducing endogenous tau ameliorates amyloid beta-induced deficits in an Alzheimer's disease mouse model

Erik D. Roberson, Lennart Mucke
2006-10-01

Acute impairment of mitochondrial trafficking by beta-amyloid peptides in hippocampal neurons
2003-10-01

Glutamate and amyloid beta-protein rapidly inhibit fast axonal transport in cultured rat hippocampal neurons by different mechanisms,Glutamate and Amyloid β-Protein Rapidly Inhibit Fast Axonal Transport in Cultured Rat Hippocampal Neurons by Different Mechanisms
2001-05-01

Mitochondrial abnormalities in Alzheimer's disease

George Perry, Massimo Tabaton
1998-11-01

Overexpression of tau protein inhibits kinesin-dependent trafficking of vesicles, mitochondria, and endoplasmic reticulum: implications for Alzheimer's disease

Article - wd:Q28585545

Welcome to Inventaire

the library of your friends and communities

you are offline