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Using induced pluripotent stem cells to investigate cardiac phenotypes in Timothy syndrome

autor

Ricardo E Dolmetsch

Joachim Hallmayer

opublikowano w: Nature

data publikacji: 2011-02-09

język: język angielski

główny temat: linia komórkowa, Timothy syndrome, fenotyp

Cites articles 27

Data

Tytuł

Autorzy

2010-07-21

Patient-specific induced pluripotent stem-cell models for long-QT syndrome

Alessandra Moretti, Milena Bellin, Lorenz Bott-Flügel, Alexander Goedel, Daniel Sinnecker
2009-11-02

Roscovitine binds to novel L-channel (CaV1.2) sites that separately affect activation and inactivation

Keith S Elmslie
2009-02-12

Functional cardiomyocytes derived from human induced pluripotent stem cells

Timothy J. Kamp
2009-08-01

Wnt3a-induced mesoderm formation and cardiomyogenesis in human embryonic stem cells
2008-12-15

The Timothy syndrome mutation of cardiac CaV1.2 (L-type) channels: multiple altered gating mechanisms and pharmacological restoration of inactivation

Keith S Elmslie
2008-11-10

Proarrhythmic defects in Timothy syndrome require calmodulin kinase II.,Proarrhythmic defects in Timothy Syndrome require calmodulin kinase II
2008-06-01

Mechanisms of cardiac arrhythmias and sudden death in transgenic rabbits with long QT syndrome

Gideon Koren
2008-02-14

Generation of pluripotent stem cells from adult mouse liver and stomach cells

Shin'ya Yamanaka, Kazutoshi Takahashi, Tsutomu Chiba
2008-02-04

The Timothy syndrome mutation differentially affects voltage- and calcium-dependent inactivation of CaV1.2 L-type calcium channels
2008-01-01

Clinical practice. Long-QT syndrome
2007-12-26

Mutation of an A-kinase-anchoring protein causes long-QT syndrome

Michael J Ackerman
2007-11-30

Induction of pluripotent stem cells from adult human fibroblasts by defined factors

Shin'ya Yamanaka, Kazutoshi Takahashi
2007-12-21

Induced pluripotent stem cell lines derived from human somatic cells
2007-08-13

Roscovitine, a cyclin-dependent kinase inhibitor, affects several gating mechanisms to inhibit cardiac L-type (Ca(V)1.2) calcium channels
2007-07-01

TRIC channels are essential for Ca2+ handling in intracellular stores

Kazuhiro Mio, Zui Pan, Chikara Sato, Hiroshi Takeshima
2005-08-01

Genetics of acquired long QT syndrome

Dan Roden
2005-04-29

Severe arrhythmia disorder caused by cardiac L-type calcium channel mutations

Frank B. Sachse, Alan H. Beggs, Katherine Timothy
2004-10-01

Ca(V)1.2 calcium channel dysfunction causes a multisystem disorder including arrhythmia and autism

Helen Tager-Flusberg, Niels Decher, Peter J. Schwartz, Carlo Napolitano, Katherine Timothy, Igor Splawski, Raffaella Bloise, Silvia Priori
2003-06-05

Human embryonic stem cells develop into multiple types of cardiac myocytes: action potential characterization
2002-02-01

Cardiotoxicity of new antihistamines and cisapride
2001-11-01

Sudden death due to cardiac arrhythmias.
2000-12-01

Functional embryonic cardiomyocytes after disruption of the L-type alpha1C (Cav1.2) calcium channel gene in the mouse

Joerg Striessnig, Robert Feil
1998-06-01

Embryonic lethality and abnormal cardiac myocytes in mice lacking ryanodine receptor type 2

Hiroshi Takeshima
1997-01-01

Biochemical and cellular effects of roscovitine, a potent and selective inhibitor of the cyclin-dependent kinases cdc2, cdk2 and cdk5

James Chong, J. Julian Blow, Jean-Guy Delcros
1996-07-01

Formation of junctions involved in excitation-contraction coupling in skeletal and cardiac muscle

Bernhard E Flucher
1996-01-01

The long QT syndrome. A review of recent molecular genetic and physiologic discoveries
1984-01-01

Ion channels in cardiac cell membranes,Ion Channels in Cardiac Cell Membranes

Artykuł - wd:Q30499663

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