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Linking the transcriptional profiles and the physiological states of Mycobacterium tuberculosis during an extended intracellular infection

author

Diogo F T Veiga

Gábor Balázsi

David G Russell

published in: PLOS Pathogens

date of publication: 2012-06-21

main subject: Mycobacterium tuberculosis, tuberculosis

Cites articles 89

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2012-02-02

Phagosomal rupture by Mycobacterium tuberculosis results in toxicity and host cell death

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2011-12-15

Asymmetry and aging of mycobacterial cells lead to variable growth and antibiotic susceptibility

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2011-03-14

aprABC: a Mycobacterium tuberculosis complex-specific locus that modulates pH-driven adaptation to the macrophage phagosome

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2011-03-01

Mycobacterium tuberculosis and the intimate discourse of a chronic infection

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Cholesterol is not an essential source of nutrition for Mycobacterium tuberculosis during infection

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2011-07-18

An ArsR-like transcriptional factor recognizes a conserved sequence motif and positively regulates the expression of phoP in mycobacteria

Chun-hui Gao, Min Yang
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DosS responds to a reduced electron transport system to induce the Mycobacterium tuberculosis DosR regulon

Prabagaran Narayanasamy
2010-09-24

Interactions of attenuated Mycobacterium tuberculosis phoP mutant with human macrophages

Olivier Neyrolles, Brigitte Gicquel, Carlos Martín
2010-07-01

Mycobacterium tuberculosis wears what it eats

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Functional genetic diversity among Mycobacterium tuberculosis complex clinical isolates: delineation of conserved core and lineage-specific transcriptomes during intracellular survival

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2010-05-03

Gluconeogenic carbon flow of tricarboxylic acid cycle intermediates is critical for Mycobacterium tuberculosis to establish and maintain infection

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The Mycobacterium tuberculosis DosR regulon assists in metabolic homeostasis and enables rapid recovery from nonrespiring dormancy
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Role of the transcriptional regulator RamB (Rv0465c) in the control of the glyoxylate cycle in Mycobacterium tuberculosis
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Mycobacterium tuberculosis is able to accumulate and utilize cholesterol

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Mycobacterium tuberculosis WhiB3 maintains redox homeostasis by regulating virulence lipid anabolism to modulate macrophage response
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Mycobacterium requires an all-around closely apposing phagosome membrane to maintain the maturation block and this apposition is re-established when it rescues itself from phagolysosomes
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Hypoxia: a window into Mycobacterium tuberculosis latency
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Studies of a ring-cleaving dioxygenase illuminate the role of cholesterol metabolism in the pathogenesis of Mycobacterium tuberculosis

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2008-06-01

Functional characterization of a vitamin B12-dependent methylmalonyl pathway in Mycobacterium tuberculosis: implications for propionate metabolism during growth on fatty acids

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Mycobacterial persistence requires the utilization of host cholesterol
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Article - wd:Q34318215

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