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The impact of p53 on the early stage replication of retrovirus.

author

Binshan Shi

published in: Virology Journal

date of publication: 2017-08-09

language: English

Cites articles 36

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Title

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p53-derived host restriction of HIV-1 replication by protein kinase R-mediated Tat phosphorylation and inactivation
2013-09-30

p21-mediated RNR2 repression restricts HIV-1 replication in macrophages by inhibiting dNTP biosynthesis pathway

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2012-06-15

Viral oncoprotein LMP1 disrupts p53-induced cell cycle arrest and apoptosis through modulating K63-linked ubiquitination of p53

Qian Tao
2011-11-06

HIV-1 restriction factor SAMHD1 is a deoxynucleoside triphosphate triphosphohydrolase

Michelle Webb, Evangelos Christodoulou, David Goldstone, Gillian I. Rice, Jonathan P. Stoye, Yanick J. Crow, Geoff Kelly, Luiz Pedro S de Carvalho
2011-10-15

Dissection of cell context-dependent interactions between HBx and p53 family members in regulation of apoptosis: a role for HBV-induced HCC

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2011-07-01

The role of unintegrated DNA in HIV infection

Mark Wainberg
2011-06-30

SAMHD1 is the dendritic- and myeloid-cell-specific HIV-1 restriction factor counteracted by Vpx

Nicoletta Casartelli, Olivier Schwartz
2011-04

CD4+ T cells from elite controllers resist HIV-1 infection by selective upregulation of p21

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2010-11-15

Transcriptional regulation of HIV-1 gene expression by p53

Pier Paolo Claudio, Luis Del Valle
2008-12-11

The common mechanisms of transformation by the small DNA tumor viruses: The inactivation of tumor suppressor gene products: p53.
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Identification and characterization of transmitted and early founder virus envelopes in primary HIV-1 infection

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P53 in cytoplasm may enhance the accuracy of DNA synthesis by human immunodeficiency virus type 1 reverse transcriptase
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Retrovirus vector production and transduction: modulation by the cell cycle
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Integration of interferon-alpha/beta signalling to p53 responses in tumour suppression and antiviral defence

Dagmar Stoiber
2003-09-01

Roles of host cell factors in circularization of retroviral dna

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Mutations in the RNase H domain of HIV-1 reverse transcriptase affect the initiation of DNA synthesis and the specificity of RNase H cleavage in vivo

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Diminished human immunodeficiency virus type 1 reverse transcription and nuclear transport in primary macrophages arrested in early G(1) phase of the cell cycle
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Mutant p53 forms a complex with Sp1 on HIV-LTR DNA.
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Article - wd:Q40088889

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