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c-Myc-induced apoptosis in fibroblasts is inhibited by specific cytokines.

published in: The EMBO Journal

date of publication: 1994-07-01

language: English

main subject: apoptotic process

Cites articles 50

Date

Title

Authors

1993-04-01

Myc-mediated apoptosis is blocked by ectopic expression of Bcl-2
1993-02-01

The role of c-myc in cell growth
1993-08-27

Bcl-2 heterodimerizes in vivo with a conserved homolog, Bax, that accelerates programmed cell death

Stanley J. Korsmeyer
1993-08-27

bcl-x, a bcl-2-related gene that functions as a dominant regulator of apoptotic cell death
1993-08-01

Characterization of A1, a novel hemopoietic-specific early-response gene with sequence similarity to bcl-2
1993-12-01

The c-Myc protein induces cell cycle progression and apoptosis through dimerization with Max
1993-10-01

Programmed cell death and the control of cell survival: lessons from the nervous system
1993-04-15

MCL1, a gene expressed in programmed myeloid cell differentiation, has sequence similarity to BCL2
1993-01-01

Oncogenic activity of the c-Myc protein requires dimerization with Max

Bruno Amati
1993-03-01

Apoptosis

J. John Cohen
1992-01-01

Max: functional domains and interaction with c-Myc

Gregory J. Kato
1992-04-01

Induction of apoptosis in fibroblasts by c-myc protein

Linda Z Penn
1992-08-01

The adenovirus E1A proteins induce apoptosis, which is inhibited by the E1B 19-kDa and Bcl-2 proteins

Eileen White, Stanley J. Korsmeyer, David Hockenbery
1992-10-01

Cooperative interaction between c-myc and bcl-2 proto-oncogenes
1992-10-01

Apoptotic cell death induced by c-myc is inhibited by bcl-2

Douglas R. Green
1992-10-01

Transcriptional activation by the human c-Myc oncoprotein in yeast requires interaction with Max.
1992-10-01

Interleukin 3 protects murine bone marrow cells from apoptosis induced by DNA damaging agents

Jacqueline Marvel
1992-06-01

The 19-kilodalton adenovirus E1B transforming protein inhibits programmed cell death and prevents cytolysis by tumor necrosis factor alpha,The 19-Kilodalton Adenovirus E1B Transforming Protein Inhibits Programmed Cell Death and Prevents Cytolysis by Tumor Necrosis Factor α
1992-01-01

The double life of the IGF-1 receptor
1992-10-01

bcl-2 gene transfer increases relative resistance of S49.1 and WEHI7.2 lymphoid cells to cell death and DNA fragmentation induced by glucocorticoids and multiple chemotherapeutic drugs
1992-09-01

Max and c-Myc/Max DNA-binding activities in cell extracts
1991-03-08

Max: a helix-loop-helix zipper protein that forms a sequence-specific DNA-binding complex with Myc
1991-01-01

The MYC protein activates transcription of the alpha-prothymosin gene

Martin Eilers
1991-01-11

Methylation-sensitive sequence-specific DNA binding by the c-Myc basic region
1991-05-01

Association of Myn, the murine homolog of max, with c-Myc stimulates methylation-sensitive DNA binding and ras cotransformation,Association of Myn, the murine homolog of Max, with c-Myc stimulates methylation-sensitive DNA binding and ras cotransformation

Edward B Ziff
1991-10-01

Constitutive c-myc expression in an IL-3-dependent myeloid cell line suppresses cell cycle arrest and accelerates apoptosis
1991-07-01

C-myc and the yeast transcription factor PHO4 share a common CACGTG-binding motif

Padma-Sheela Jayaraman
1990-11-01

Sequence-specific DNA binding by the c-Myc protein
1990-11-01

An amino-terminal c-myc domain required for neoplastic transformation activates transcription
1990-08-01

Complementation by BCL2 and C-HA-RAS oncogenes in malignant transformation of rat embryo fibroblasts

Carlo Maria Croce
1990-04-01

Negative autoregulation of c-myc transcription
1990-09-01

Role of the two activating domains of the oestrogen receptor in the cell-type and promoter-context dependent agonistic activity of the anti-oestrogen 4-hydroxytamoxifen

Daniel Metzger

Article - wd:Q40792572

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