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Mitochondrial Aging Defects Emerge in Directly Reprogrammed Human Neurons due to Their Metabolic Profile

author

Larissa Traxler

Frank Edenhofer

Baptiste N Jaeger

1985 -

Jerome Mertens

Fred Gage

1950 -

Apua C M Paquola

Jürgen Winkler

Leah F Boyer

2000 -

mű, amelyben megjelent: Cell Reports

date of publication: 2018-05-01

Cites articles 38

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2017-11-02

Direct Reprogramming Rather than iPSC-Based Reprogramming Maintains Aging Hallmarks in Human Motor Neurons
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Age-Related Accumulation of Somatic Mitochondrial DNA Mutations in Adult-Derived Human iPSCs

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Parkin and PINK1 Patient iPSC-Derived Midbrain Dopamine Neurons Exhibit Mitochondrial Dysfunction and α-Synuclein Accumulation

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Directly Reprogrammed Human Neurons Retain Aging-Associated Transcriptomic Signatures and Reveal Age-Related Nucleocytoplasmic Defects

Jun Yao, Jerome Mertens, Emily Hatch, José Tiago Gonçalves, Fred Gage, Apua C M Paquola, Jürgen Winkler
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Computational classification of mitochondrial shapes reflects stress and redox state
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Disease-causing mitochondrial heteroplasmy segregated within induced pluripotent stem cell clones derived from a patient with MELAS.

Timothy J. Nelson
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Motile axonal mitochondria contribute to the variability of presynaptic strength
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Tissue- and cell-type-specific manifestations of heteroplasmic mtDNA 3243A>G mutation in human induced pluripotent stem cell-derived disease model

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Presynaptic mitochondrial morphology in monkey prefrontal cortex correlates with working memory and is improved with estrogen treatment

Yuko Hara
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Human iPSC-based modeling of late-onset disease via progerin-induced aging

Lorenz Studer, Pankaj K Mandal, Robert L Bowman
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Induced pluripotent stem cells generated from diabetic patients with mitochondrial DNA A3243G mutation
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The axonal transport of mitochondria
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Oxidative phosphorylation, not glycolysis, powers presynaptic and postsynaptic mechanisms underlying brain information processing

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Fusion and fission: interlinked processes critical for mitochondrial health
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The striatum is highly susceptible to mitochondrial oxidative phosphorylation dysfunctions
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Rejuvenating senescent and centenarian human cells by reprogramming through the pluripotent state

Jean-Marc Lemaitre, John De Vos, Sylvain Lehmann
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Mitochondrial-associated cell death mechanisms are reset to an embryonic-like state in aged donor-derived iPS cells harboring chromosomal aberrations

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Maria Manczak

Article - wd:Q88920896

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